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DNP acts as a proton ionophore in the mitochondria, shuttling protons across membranes and dissipating the proton gradient that drives ATP synthesis. This causes ATP production to become uncoupled from electron transport, resulting in the energy from respiration being lost as heat instead of being stored in ATP. DNP was historically used as a weight loss drug due to its effects of increasing metabolic rate and oxygen consumption, but it is very dangerous and can cause hyperthermia due to the unregulated hea

Author

SkenzKenz

Language

EN