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This review discusses the phenomenon of nitrate tolerance associated with nitroglycerin, a common antianginal agent, which diminishes in effectiveness over time due to neurohormonal activation and intrinsic vascular changes. The mechanisms behind this tolerance involve increased oxidative stress and impaired bioactivation of nitroglycerin, leading to reduced responsiveness of vascular smooth muscle. The review also explores the different pathways of nitroglycerin bioactivation and their implications for tre

Author

badymore

Language

EN