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This study investigates the role of IL-6 signaling in enhancing the susceptibility of castration-resistant prostate cancer (CRPC) cells to natural killer (NK) cell-mediated cytotoxicity by altering PD-L1 and NKG2D ligand levels. IL-6-knockdown CRPC cells showed increased NK cell susceptibility compared to control cells, and inhibition of JAK/Stat3 signaling further enhanced this effect. These findings suggest that targeting IL-6 signaling may improve NK cell-mediated immune responses in CRPC, offering poten

Author

Macarena Fernández Villarroel

Language

EN