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This review article discusses the molecular and cellular mechanisms involved in the pathogenesis of coronary artery ectasia (CAE), a condition characterized by the abnormal dilatation of coronary arteries. It highlights the coexistence of CAE with coronary artery disease (CAD) and the implications for clinical management and treatment strategies. Understanding the underlying etiology of CAE may improve treatment modalities and provide insights into its clinical significance.

Author

4yjmhppmhm

Language

EN