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This document discusses the potential of oral lumbrokinase (DLBS 1033) as a fibrinolytic drug. It summarizes research showing that DLBS 1033 increases autolytic and fibrinolytic activity, inhibits platelet aggregation, reduces proinflammatory factors, and improves blood flow in clinical studies of ischemic stroke, atherosclerosis, and angina patients. The document concludes that due to its antithrombotic, antiplatelet, and fibrinolytic mechanisms, lumbrokinase presents a challenging opportunity as a fibrino

Author

faisalsyahril

Language

EN