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This study identifies four distinct subpopulations of adipose progenitor cells (APCs) in human visceral adipose tissue, with a significant increase in the CD9+CD55low subset in patients with type 2 diabetes (T2D). These pathogenic APCs contribute to glycemic disturbances by producing bioactive proteins that create a detrimental environment, leading to increased lipolysis in adipocytes. The findings suggest that targeting APCs may offer a new therapeutic approach to combat T2D and obesity-related metabolic d

Author

Zhongqi Zhao

Language

EN