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This study investigates the cardio-protective effects of exercise training against cardiac hypertrophy and the duration of these effects after cessation of exercise. Results show that exercise provides significant protection for at least two weeks post-training, primarily through the activation of endothelial nitric oxide synthase (eNOS). The findings suggest that eNOS signaling plays a crucial role in sustaining the cardio-protective phenotype following exercise cessation.

Author

k

Language

EN