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This minireview discusses the reactivation of Epstein–Barr virus (EBV) by various biological agents, including bacteria and viruses, particularly under immunocompromised conditions or stress stimuli. It highlights how certain pathogens, such as Aggregatibacter actinomycetemcomitans and Human simplex virus-1, can induce EBV reactivation through specific molecular mechanisms. The review aims to shed light on the gaps in research regarding EBV reactivation and its implications for disease severity and manageme

Author

Jorge Estrada

Language

EN