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The study investigates the impact of cytochrome c oxidase (COX) defects on cellular metal homeostasis, particularly focusing on copper, iron, manganese, and zinc levels in Drosophila melanogaster models. Findings reveal that COX deficiency leads to altered metal distribution, with decreased copper in mitochondria and increased levels in the cytosol, alongside changes in the expression of metal transporters and metallothioneins. The research indicates that COX defects contribute to mitochondrial disease by d

Author

ncehilda

Language

EN