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This study investigates the phagocytosis of ultrasound contrast agent microbubbles by Kupffer cells in the liver and its impact on delayed parenchymal phase imaging. The results show that Kupffer cells effectively phagocytosed certain contrast agents, with 99% of SonazoidTM and OptisonTM being taken up, while the elimination of Kupffer cells reduced the intensity of delayed images. These findings suggest that Kupffer cells play a significant role in the delayed imaging observed in contrast-enhanced ultrasou

Author

haichenhu416

Language

EN