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This study investigates the effects of metformin on stress-induced premature senescence (SIPS) in human umbilical vein endothelial cells (HUVECs) induced by hydrogen peroxide. Metformin was found to alleviate SIPS by enhancing cell migration and angiogenesis, regulating mitochondrial function, and promoting migrasome-mediated mitocytosis. These findings suggest that metformin could serve as a therapeutic agent for vascular dysfunction associated with SIPS.

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phuongndk25408a

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EN