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This document contains an NIH R01 grant proposal submitted in response to a funding opportunity. The proposal focuses on investigating how mitochondrial DNA (mtDNA) damage and polymorphisms modify neonatal lung injury caused by oxidative stress, which can lead to bronchopulmonary dysplasia (BPD). The proposal has 3 specific aims: 1) evaluate how mtDNA damage amplifies lung injury, 2) identify how mtDNA haplotype influences lung injury, and 3) define biomarkers for BPD risk based on platelet mtDNA. The propo

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[email protected]

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EN