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Salidroside Inhibits Ischemia/Reperfusion-Induced Myocardial Apoptosis by Targeting Mir-378a-3p Via the Igf1r/Pi3k/Akt Signaling Pathway 1 Materials and Methods 2 Reagents 2 H/R Injury 2 Cell Transfection 2 RT-qPCR Assay 2 Lactate Dehydrogenase Enzyme Assay 2 MTT Assay 2 Flow Cytometry Analysis 2 Measurement of Mitochondrial Membrane Potential 2 Hoechest Staining 4 I/R Model and Drug Treatment 4 Masson Staining 4 ELISA Assay 4 Western Blot 4 TUNEL Staining 4 Statistical Analysis 4 Results 4 SAL Reduced Apoptosis in H9c2 Cells Stimulated With H/R 4 SAL Inhibited Mitochondrial ROS Production in H9c2 Cells Stimulated With H/R 6 SAL Restored Mitochondrial Membrane Potential in H9c2 Cells Stimulated With H/R 6 SAL Attenuated Cardiac Injury in the I/R Rats 6 SAL Alleviated Myocardial Apoptosis in the I/R rats 7 SAL Upregulated miR-378a-3p to Inhibit Cell Apoptosis in H9c2 Cells Stimulated With H/R 10 SAL Upregulated miR-378a-3p via IGF1R/PI3K/Akt Signaling Pathway in H9c2 Cells Stimulated With H/R 12 Discussion 12 Conclusions 13 Acknowledgments 13 References 13

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UND

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nonfiction