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This study investigates the origins and maintenance mechanisms of resident arterial macrophages, revealing that they arise from embryonic CX3CR1+ precursors and postnatally from bone marrow-derived monocytes. The research shows that in adulthood, arterial macrophages are sustained through local proliferation rather than monocyte recruitment, and their survival is dependent on the CX3CR1-CX3CL1 axis. The findings highlight the distinct gene expression profiles of arterial macrophages compared to other macrop

Author

r9gm2ttmnr

Language

EN