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This document summarizes evidence regarding whether altered uric acid metabolism contributes to diabetic kidney disease pathophysiology. It reviews how uric acid is generated in diabetes and animal/cell studies showing uric acid causes endothelial dysfunction, oxidative stress, and inflammation. Clinical studies associate higher uric acid with increased risk of chronic kidney disease and end-stage renal disease. While relationships are seen, whether uric acid directly causes kidney damage or is a marker is

Author

Nazihan Safitri Alkatiri

Language

EN