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The document discusses the important role of lipoprotein(a) or Lp(a) in cardiovascular disease risk. Lp(a) consists of apolipoprotein B and apolipoprotein A, inheriting atherogenic traits from both. Recent evidence from genetic studies shows that high Lp(a) levels causally increase risk of various cardiovascular diseases. New therapies targeting Lp(a) lowering such as PCSK9 inhibitors and antisense oligonucleotides have shown promise in reducing residual cardiovascular risk.

Author

Physics Tution

Language

EN